Screw bananas and oatmeal and dietary “have-to”

Here’s what I have to finally say: drop the damn banana, skip the “healthy” oatmeal, put down the whole grain, seed-filled cardboard-like toast you’ve convinced yourself you prefer to plain white bread, AND go ahead and skip breakfast if you want to. Don’t have time for lunch? Don’t stress about it, eat when you’re actually calm and hungry and have time to enjoy and focus on eating.

The long and the short of the sciencey bits are as follows: (this stuff lights me up and excites me way more than your average Josephine and to save your eyes from rolling back in your head I’ll keep this brief and bullet pointy) There is SO MUCH more to say, so look for future posts on the subject.

Diabetes= carbohydrate toxicity

What happens when you eat carbohydrate? Here’s what happens after you swallow:
All carbohydrates (rice, bread, chocolate, sugar, pasta, corn, apple…doesn’t matter) are turned into glucose and fructose and galactose (ie, SUGAR) by the time they hit the bloodstream.

Glucose quickly stimulates insulin response (insulin= fat storage hormone) and some is stored in the liver and muscle tissue as glycogen, the fuel form of glucose. Only about 2-3k calories of which are able to be stored at any given time in the body. The rest is converted to fat and stored that way. At any given time, even a fit individual, has about 40,000 calories of readily available energy stored up. WHA!?!? Yea. I’d much rather use up my saddlebag fat for power than the quick burning, measly 3k of sugar from my liver, but that’s me.

Fructose doesn’t impact insulin the way that glucose does. Without the stimulation of insulin the fructose (think processed foods, high fructose corn syrup, etc.) stays in the blood stream, rapidly raising blood sugar and wreaking inflammatory, circulatory havoc. It is exclusively processed in the liver (same as alcohol and other toxins) where it is converted into triglycerides, VLDL and LDL cholesterol. Yup. Bad cholesterol isn’t consumed, it is created from sugar by the liver.

Fat Cells= energy reserves

Between meals is the time when fat is used as your energy source. (go ahead and allow yourself the pleasure of skipping obligatory meal times if you’re not hungry! It’s called Intermittent Fasting which hosts an enormous number of benefits from reduction of inflammation to reducing cancer markers…)

Carbohydrate is NOT an essential nutrient, you do not need to consume it. The body has efficient, natural processes in place that turn amino acids (found in meet, fat, etc.) into the necessary small amount of glucose. YOU DO NOT NEED TO EAT CARBS/SUGAR to survive or function optimally. Period. (if you’re interested, the process is called gluconeogenisis)

On Oatmeal:
Oatmeal does in fact follow through on Quaker Oats’ claim, it does remove cholesterol from the system. Fiber absorbs cholesterol IN THE GUT and you poop it out.

It DOES NOT lower BLOOD cholesterol. Your doc doesn’t test how much cholesterol is in your gut, they measure your BLOOD cholesterol because that’s the stuff that’ll kill ya. Pooping out dietary cholesterol (which is primarily “good cholesterol”, HDL)* while, as discussed above, turning the 27-50+ grams of carbohydrate in your morning bowl of oats into…wait for it…glucose which is then converted into LDL, “bad cholesterol”, stored as body fat AND turns your body into a sugar processing machine instead of a fat burning one.

So, put down the friggin’ oatmeal please, don’t buy the hype. Love you!!

xoxox
C

*very very little LDL exists in the foods we eat, though one could make an argument that our grain-fed, undernourished, sick, stressed, factory-farmed animal products are likely not as healthy a source of HDL as they once were…

Resources:
Silva, Pedro. “The Chemical Logic Behind Gluconeogenesis”
Elliott SS, Keim NL, Stern JS, Teff K, Havel PJ (2002). “Fructose, weight gain, and the insulin resistance syndrome”, http://ajcn.nutrition.org/content/76/5/911.full
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2682989/
Very high fructose intake increases serum LDL-cholesterol and total cholesterol: a meta-analysis of controlled feeding trials.
Zhang YH1, An T, Zhang RC, Zhou Q, Huang Y, Zhang J.,
http://www.ncbi.nlm.nih.gov/pubmed/23825185

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